• 综述 •
金卫涛, 杨婷, 贾吉美, 周晓斐. 基于纳米材料介导的自噬治疗癌症[J]. 化学进展, 2023, 35(11): 1655-1673.
Jin Weitao, Yang Ting, Jia Jimei, Zhou Xiaofei. Nanomaterials-Mediated Autophagy-Based Cancer Treatment[J]. Progress in Chemistry, 2023, 35(11): 1655-1673.
随着纳米技术的快速发展,纳米材料在癌症治疗领域得到了广泛的应用。众所周知,自噬作为一个维持细胞稳态的过程,在癌症发展中发挥着促生存和促死亡的双重作用。癌细胞中的自噬水平远高于正常细胞,从而导致各种治疗策略效果不理想。通过扰动自噬协同治疗癌症已经成为了一种可行的方案,但是传统自噬扰动剂如氯喹等可能导致某些其他的副作用。此外,已有研究证明了纳米材料可以作为一种新型的自噬扰动剂,但是纳米材料干扰自噬的机制需要更加深入的了解。本文综述了癌症与自噬的双重关系,并重点介绍了多种纳米材料通过扰动自噬诱导癌细胞死亡或凋亡,或者通过扰动自噬增强癌细胞对传统癌症治疗的敏感性,以及它们调控自噬的机制。
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NMs | Size (nm) | shape or dispersity | Coating | Drug or therapies | Model cells | Mechanism | Autophagy | Effect | ref |
---|---|---|---|---|---|---|---|---|---|
Au NPs | 20 | sphere | PEG | immunotherapy | Hepa1-6 cells; RAW 264.7 cells | Lysosome alkalization; membrane permeabilization | Inhibition | Increased sensitivity | |
Au NPs | 30~60 | Peanut | Bare | — | SKOV-3 cell | ROS upregulation | Inhibition | Apoptosis | |
Ag NPs | 66.92 | sphere | Bare | — | PC-3 cell | lysosome injury; cell hypoxia | Inhibition | Cell death | |
Fe3O4-Au NPs | 15~25 | sphere | — | DOX | HepG2 cells | Enhancing autophagosome formation | Induction | Reduce drug resistance | |
Ag NPs | 59 | sphere | Bare | — | HT-29 cells | JNK activation and eIF2α phosphorylation | Induction | Apoptosis | |
Fe@Au NPs | — | core-shell structure | — | — | OECM1 cell | Mitochondria damage | Inhibition | Cell death | |
Ag NPs | 26.5 | sphere | PVP | — | HeLa cell | PtdIns3K-dependent | Induction | Ehances the anticancer activity | |
Fe3O4 NPs | 36 | sphere | PEG | PTX | U251 cell | ROS upregulation | Induction | Reduce drug resistance | |
Ag NPs | 13 | sphere | — | — | A549 cell | ROS upregulation | Induction | Apoptosis | |
Ag NPs | 8 | sphere | protein | Cisplatin | OS cell; HCC cell | MAPK pathways | Induction | Reduce drug resistance | |
Fe2O3 NPs | 10 | sphere | DMSA | — | SK-Hep-1 cell | ROS upregulation; MAPK pathways | Induction | Cell death | |
CuO NPs | 10 | sphere | — | — | MCF7 cell | ROS dependent | Induction | Growth inhibition | |
ZnO NPs | 21 | — | — | Sorafenib | Huh 7 cell | Promoting p53 Gene | Induction | Apoptosis | |
ZnO NPs | 63 | — | — | — | MCF7 cell | — | Inhibition | Apoptosis | |
SiO2 NPs | 86 | sphere | HCT-116 cells | ER stress | Induction | Cell survival | |||
Ag NPs | 15.38 | sphere | PVP | Radiotherapy | U251 cells | ROS upregulation | Induction | Increased sensitivity | |
IONPs | 37 | — | PEG | — | U251 cells | Beclin 1/ATG 5 pathways | Induction | Ferroptosis | |
Gd2O3 NPs | — | — | — | Cisplatin | HeLa cells | — | Inhibition | Reduce drug resistance | |
SiO2 NPs | 125 | sphere | — | Propranolol | HemSCs cells | ER stress | Induction | Cell death | |
ZnO NPs | 20 | — | — | — | SKOV3 cells | ROS upregulation | Induction | Apoptosis | |
SiO2 NPs | 198 | sphere | PDA; PEG | DOX | MCF7 cells | AKT-mTOR-p70S6K pathway | Induction | Cell death | |
ND | 191 | — | — | Hypoxia | HeLa cells; MCF7 cells | — | Inhibition | Apoptosis | |
ZnO NPs | — | — | — | Cisplatin | SGC7901 cells;BGC823 cells | — | Inhibition | Reduce drug resistance | |
GO | 450 | sheet | DMSO | Cisplatin | Skov-3 cells | — | Induction | Cell death | |
DWCNTs | — | Tube | — | — | DHD/K12/Trb cell line | Intracellular acidification | Induction | Cell death | |
Se NPs | 70 | Amorphous solid | — | Astragalus Polysaccharides | MCF7 cells | ROS upregulation and Mitochondria damage | Inhibition | Apoptosis | |
BPQDs | 140 | Monodisperse | Platelet membrane | Hederagenin | MCF7 cells; RAW 264.7 cells | ROS upregulation and Mitochondria damage | Induction | Apoptosis | |
Co3O4 NPs | 200 | sphere | — | Photothermal therapy | U-87 MG cells | Llysosomal function damage | Blockage of autophagic flux | Cell death |
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